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Table 1 Demographics of study population

From: Integrative network analysis reveals different pathophysiological mechanisms of insulin resistance among Caucasians and African Americans

Trait

Caucasian or European-American (CA)

African-American (AA)

N

99

37

Gender (M/F)

42/57

21/16

Age (Yrs)

39.7 ± 10.9

42.1 ± 9.1

WHR

0.89 ± 0.19

0.89 ± 0.07

BMI (kg/m2)

28.2 ± 4.9

29.5 ± 6.3

% Fat mass

32.6 ± 9.0

29.6 ± 10.9

Triacylglycerol (mmol/l)

1.45 ± 1.36

1.14 ± 0.71

Total Cholesterol (mmol/l)

4.75 ± 0.92

4.47 ± 1.01

LDL Cholesterol (mmol/l)

2.75 ± 0.76

2.56 ± 0.89

HDL cholesterol (mmol/l)

1.39 ± 0.47

1.38 ± 0.33

Fasting Glucose (mmol/l)

4.74 ± 0.46

4.72 ± 0.53

2 h Glucose (mmol/L) ¶

5.80 ± 1.63

5.51 ± 1.91

Fasting Insulin (pmol/l)

43.6 ± 39.8

46.6 ± 61.2

HOMA-IR ¶

1.36 ± 1.34

1.50 ± 2.35

Matsuda Index ¶

7.60 ± 5.48

9.18 ± 6.12

SI (×10−4.min−1 [μU/ml]−1)*

3.8 ± 2.3

3.4 ± 1.9

AIRG (pmol/L)*

2860 ± 2177

4586 ± 3148

DI*

1515 ± 1144

2061 ± 1257

  1. Continuous variables are shown as mean ± SD. ¶Metabolic measurements from OGTT; *Metabolic traits from MINMOD analysis of FSIGT evaluation of non-diabetic individuals. BMI, Body mass index; WHR, waist to hip ratio; LDL, Low density lipoprotein; HDL, High Density lipoprotein; HOMA-IR, homeostatic model assessment of insulin resistance; SI, insulin sensitivity index; AIRG, Acute insulin response to glucose; DI, Disposition index. Units are taken from MINMOD program.