Fig. 6From: Differential alternative splicing regulation among hepatocellular carcinoma with different risk factorsAlternative splicing events of IP6K2. a Schematic of skipping exon 3 on the IP6K2 transcript, b PSI level of ES of IP6K2 in the three different etiology groups (HBV, HCV, and Alcohol groups). IP6K2, inositol hexakisphosphate kinase 2; ES, exon skipping; PSI, percent spliced in; HBV, hepatitis B virus; HCV, hepatitis C virusBack to article page