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Fig. 1 | BMC Medical Genomics

Fig. 1

From: The interactions between dietary fats intake and Caveolin 1 rs 3807992 polymorphism with fat distribution in overweight and obese women: a cross-sectional study

Fig. 1Fig. 1Fig. 1Fig. 1Fig. 1Fig. 1

Interaction between rs 3807992 genotypes and dietary fats on the mean some of fat distribution variables. a–d With increasing total fat intake with two risk alleles (A), 0.11 cm2 vaisceral fat level (p-interaction: 0.86), 0.02 kg trunk fat (p-interaction: 0.76), 0.09 cm WC (p-interaction: 0.19) increased and that with increasing total fat intake with one risk alleles (A), 0.17 cm2 viseral fat level (p-interaction: 0.05) increased and 0.004 cm WHR decreased (p-interaction: 0.90). e With increasing SFA intake with having two risk alleles (A), 0.05 kg trunk fat increased (p-interaction: 0.23). f, g With increasing TFA intake with having one risk alleles (A), 5.12 cm WHR (p-interaction: 0.04) increased and 393.89 cm2 visceral fat level (p-interaction: 0.44) increased. h With increasing MUFA intake with having two risk alleles (A), 0.08 cm WHR (p-interaction: 0.28) decreased. i With increasing PUFA intake with having two risk alleles (AA), 0.06 cm2 visceral fat level (p-interaction: 0.19) increased. j With increasing linoleic acid intake with having one risk alleles (A), − 0.21 cm WHR (p-interaction: 0.03) decreased. k With increasing ALA intake with having one risk alleles (A), − 2.50 cm WHR (p-interaction: 0.06) decreased

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